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These benefits indicated that increased expression in the Ex4a(+)WT1 isoform may well contribute on the induction of apoptosis. What's more, to look at the consequences from the endogenous Ex4a(+)WT1 isoform on Dox-induced apoptosis, K562 cells were being transfected with either of two distinctive WT1 Ex4a-specific siRNAs (si-4a-1 and si-4a-2) or manage siRNA (si-control) for 24 h, dealt with with

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